Tag Archives: Kenny L de Meirleir

Evaluation of four clinical laboratory parameters for the diagnosis of ME

Posted on 02/07/2019 by wames

Evaluation of four clinical laboratory parameters for the diagnosis of myalgic encephalomyelitis, by Kenny L. De Meirleir, Tatjana Mijatovic, Krishnamurthy Subramanian, Karen A. Schlauch and Vincent C. Lombardi in Journal of Translational Medicine 2018 16:322 [Published: 21 November 2018] Research abstract: Background: Myalgic encephalomyelitis (ME) is a complex and debilitating disease that often … Continue reading →

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Nutritional modulation of the intestinal microbiota

Posted on 07/15/2018 by wames

Review abstract: Nutritional modulation of the intestinal microbiota: future opportunities for the prevention and treatment of neuroimmune and neuroinflammatory disease, by Vincent C Lombardi, Kenny L De Meirleir, Krishnamurthy Subramanian, Sam M Nourani, Ruben K Dagda, Shannon L Delaney, András Palotás … Continue reading →

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NVCBR researchers close in on a diagnostic test for ME/CFS patients

Posted on 03/10/2017 by wames

Nevada Center for Biolomedical Research news item, by Kellen Jones, 28 February 2017: NVCBR researchers close in on a diagnostic test for ME/CFS patients Physicians lack effective tools to aid in the diagnosis and treatment of those who are impacted by … Continue reading →

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Dysfunctional low affinity antibodies leads to gut & immune problems in ME/CFS

Posted on 07/05/2016 by wames

Research abstract: Myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) is a heterogeneous illness characterized by a number of comorbid conditions; gastrointestinal (GI) dysregulation make up one subgroup of this disease. IgA is the most abundant antibody isotype found in mucosal secretions including … Continue reading →

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HERV found in the guts of people with ME

Posted on 06/10/2013 by wames

Research Abstract Background: Myalgic encephalomyelitis (ME) is a debilitating illness of unknown etiology characterized by neurocognitive dysfunction, inflammation, immune abnormalities and gastrointestinal distress. An increasing body of evidence suggests that disruptions in the gut may contribute to the induction of … Continue reading →

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