Tag Archives: neuro-inflammatory

Videos: INIM ME/CFS conference 2023

Posted on 06/08/2023 by wames

Institute for Neuro-Immune Medicine Conference 2023   On May 19th in Florida USA the INIM conference, led by Dr Nancy Klimas explored the many parallels formed around COVID-19 and ME/CFS. Viral Reactivation, Mast Cell Activation, Systemic and Vascular Inflammation, and … Continue reading →

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A neuro-inflammatory model can explain the onset, symptoms and flare-ups of ME/CFS

Posted on 01/14/2020 by wames

A neuro-inflammatory model can explain the onset, symptoms and flare-ups of myalgic encephalomyelitis/chronic fatigue syndrome, by Angus Mackay in Journal of Primary Health Care 11(4) 300-307 2019 [https://doi.org/10.1071/HC19041]   Abstract: A neuro-inflammatory model is proposed to explain the onset, symptoms … Continue reading →

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Neuroimmunology: what role for autoimmunity, neuroinflammation, & small fiber neuropathy in FM, CFS, & adverse events after HPV?

Posted on 10/25/2019 by wames

Neuroimmunology: what role for autoimmunity, neuroinflammation, and small fiber neuropathy in Fibromyalgia, Chronic Fatigue Syndrome, and adverse events after Human Papillomavirus Vaccination?, by Varvara A Ryabkova, Leonid P Churilov and Yehuda Shoenfeld in Int. J. Mol. Sci. 2019, 20(20), 5164; [https://doi.org/10.3390/ijms20205164] … Continue reading →

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Abnormal cytokine/chemokine profile in ME/CFS is associated with clinical symptoms

Posted on 12/02/2015 by wames

Research highlights: Abnormal cytokine/chemokine profile in ME/CFS associated with clinical symptoms. A cluster of IL-16, IL-7 and VEGF-A that may suggests neuro-inflammation as the pathogenesis mechanism in ME/CFS. Potential diagnostic biomarkers proven by vigorous mathematical/statistical analysis. Abstract: Recently, differences in … Continue reading →

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Central pathways causing fatigue in CFS

Posted on 02/16/2015 by wames

Article abstract Background The genesis of severe fatigue and disability in people following acute pathogen invasion involves the activation of Toll-like receptors followed by the upregulation of proinflammatory cytokines and the activation of microglia and astrocytes. Many patients suffering from … Continue reading →

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